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DePaul　University

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DePaul University, Chicago IT Outsourcing Panel Discussion DePaul University CTI - Chicago, Illinois February 23, 2001 DePaul University, Chicago Russell Clark, Director Benedictine University DePaul University

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DePaul　University関連エントリー

[Newly published books]
Marcus Giaquinto, Visual Thinking in Mathematics, Oxford University Press, ￡40. Michael DePaul & Linda Zagzebski, Intellectual Virtue: Perspectives from Ethics and Epistemology, Oxford University Press, ￡18.99. Jordan Ganeri, The ...
cake!
... かなりビューティホーなお菓子を食べに行った♪ はやとさんには、Depaul Universityの近くのお店の話とか、イベントとか、駅のこととかイロイロ教えてもらってかなり参考になった ってか電車男ってまじおもしろい！！ あの主役の役者さんの名前なん ...
コンピューター・システムとチームと人脈
... Half Moon Bay Review） Danny Evans 1981 Chicago White Sox（an intern from DePaul University） 1996 Chicago White Sox assistant GM 2000 Chicago Cubs a consultant 2001 MLB a consultant/Fos Sports a television analyst ...
もう。。。
... また消えそうやし まじ、なぐんで ってか去年、これから私が行くDepaul Universityに留学いってた、はやとさんがメールくれて、明日大学で会えるかもしれへん やばい、なんか人見知りしてしまいそうで緊張 勉強とか留学とかで病んでるときとか ...
TransNews Annex : What if it's (sort of) a boy and (sort ...
... counselors, a young woman stood up to speak. A 20-year-old DePaul student, she was very pretty, in a chunky necklace, floral shirt ... he told me in early summer in his office at the University of California in San Francisco, where he’s now an ...
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DePaul　Universityに関する質問

専門用語が多すぎてうまく訳をすることができません。この英文の訳をお願いします....
専門用語が多すぎてうまく訳をすることができません。この英文の訳をお願いします。また、無料でこういった科学英語の訳をできるサイトがあれば教えてください。The avian ChB6 alloantigen triggers apoptosis in a mammalian cell line.Pifer J, Robison D, Funk PE.Department of Biological Sciences, DePaul University, Chicago, IL 60614,USA.Many developing B lymphocytes are deleted by apoptosis. However, the mechanism signaling their demise remains poorly understood. Like mammals, chicken B cells are selected during their development; >95% of the cells in the bursa of Fabricius die without entering the secondary immune system. The molecule chB6 (Bu-1) has been used as a marker to identify B cells in the chicken. ChB6 is a type I transmembrane glycoprotein whose function is enigmatic. We have provided evidence that chB6 can induce a rapid form of cell death exhibiting characteristics of apoptosis. Here we further examine cell death induced by chB6 in a transfected mouse cell line. ChB6 is shown to cause apoptosis in this cell line as detected by a TUNEL assay for DNA fragmentation. This apoptosis is subject to regulation by signals from growth factor or by Bcl-x(L). Furthermore, we show that Ab binding to chB6 leads to cleavage of caspase 8, caspase 3, and poly(ADP ribose) polymerase. Overall, these data support the hypothesis that chB6 is a novel death receptor on avian B cells.
専門用語が多すぎてうまく訳をすることができません。この英文の訳をお願いします....
専門用語が多すぎてうまく訳をすることができません。この英文の訳をお願いします。また、無料でこういった科学英語の訳をできるサイトがあれば教えてください。The avian chB6 alloantigen induces apoptosis in DT40 B cells.Funk PE, Pifer J, Kharas M, Crisafi G, Johnson A.Department of Biological Sciences, DePaul University, 2325 N. Clifton, Chicago, IL 60614, USA. pfunk@depaul.eduIn avian species, B-lymphocytes develop in the bursa of Fabricius. Cells developing in the bursa are subject to signals regulating their survival, with the majority of cells dying by apoptosis within the bursa. However, the molecules delivering the signals influencing this life and death decision remain enigmatic. We have previously shown that antibodies against the chB6 alloantigen present on avian B-lymphocytes can induce a rapid form of cell death. Here we extend this finding by showing that anti-chB6 antibodies induce true apoptosis in DT40 cells without visible membrane damage. This apoptosis results in DNA degradation and morphologic changes characteristic of apoptosis. Furthermore, this apoptosis is coincident with a loss of mitochondrial membrane potential and is inhibited by either overexpression of bcl-x(L) or the presence of inhibitors of caspase 8, 9, or 3 activity. Collectively these data argue that chB6 may function as a novel death receptor on avian B-lymphocytes and support the use of DT40 as an amenable model to study the signaling involved in chB6-induced apoptosis.
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